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These medications should not be automatically avoided in Crohns disease, and clinicians should feel more comfortable considering them when clinically indicated

73 Studies 74 have shown that TNF- can amplify local inflammation by activating the NLRP3 inflammasome and inducing IL-1 secretion, while also binding to TNF-R1 on esophageal sensory neurons and activating the p38 MAPK pathway, thereby increasing the open probability of TRPA1 channels and enhancing mucosal sensitivity to refluxate

Studies indicate positive modifications to gut microbiota in just eight weeks of use, supporting both digestive comfort and microbial diversity